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Neuroimaging modalities, such as fMRI, PET, DTI, and MRS, have consistently demonstrated functional and structural abnormalities in pain-processing networks, supporting the central sensitization model and revealing potential targets for therapeutic modulation
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Several options are available for pharmacological inhibition of glucose metabolism: glucose uptake (phloretin) (150) and glycolytic enzymes (3-bromopyruvate and Koningic acid for GAPDH) (151, 152)
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