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If we go beyond this enormous significance of Complex 1 of the mitochondrial respiratory chain for Parkinson’s and just look at what the main theory about how it happens in the first place, it’s a degeneration of the dopamine-based neurons in the basal ganglia that are controlling movement

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At the level of glutamate release, triggered by the inflammation-induced NO elevation, full excitotoxic neuronal death would be expected to occur.[186] In essence, when mitochondrial energy generation was only partially impaired, excitotoxicity was the major damaging process, however, with later total energy loss (mitochondrial collapse) necrotic death became the major death process