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Environmental factors, such as infections, pollutants, smoke and detergents, can alter the skin barrier and trigger an inflammatory response through the generation of reactive oxygen species (ROS) (19–22)

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ALA/DHLA can also maintain cellular antioxidant status indirectly by inducing the absorption of other antioxidant molecules or enhancing the synthesis of endogenous antioxidants, such as vitamin C (ascorbic acid) and, indirectly, vitamin E, or antioxidant enzymes and the ratio of reduced/oxidized glutathione (GSH/GSSG) [25] (Figure 3)