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Indeed, both Ndufs1 and Nduv1 form part of the NADH binding site and S-glutathionylation of either protein or both may actively prevent the two-electron reduction of FMN by NADH—however, prolonged S-glutathionylation may result in electron backflow from a reduced quinone pool and production of O 2 • − by FMN (Figure 3)
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